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How Cannabis Cures Cancer ?
How in the world can Banned Drug which toxicities your body and mind can cure Cancer?
To start with; if someone says Cannabis Toxicities your body and mind is Highly Offending cause there is no toxicity in it? To be more precise; the molecules ( or chemicals ) which makes any mammal high or stoned in Cannabis is known as Cannabinoids , and our Human Body and many Mammals Produce these Cannabinoids Naturally in their own body secreted by pituitary glands. Mothers Breast Milk has natural Cannabinoids there are many Rich Cancer Patients who buy Little Breast Milk from multiple donor to treat their Cancer.
So that doesn’t Mean it will Cure Cancer.
Yes , that doesn’t mean anything yet .
Medical marijuana is chemotherapy, natural style, for all cancer patients. The two forms of hemp oil, one with THC and CBD and the other CBD alone (which is pretty much legal everywhere) provide the body with chemo therapeutics without the danger and staggering side effects. There are many chapters in this book about cancer patients using marijuana but in this one we present a quick overview of the science that backs up the assertion that every cancer patient and every oncologist should put medical marijuana on their treatment maps.
What you will see in this chapter is reference to many scientific studies that are all viewable on governmental sites. The United States government is pathetic in its dishonesty about medical marijuana both believing in it and holding patents for its medical use and claiming at the same time that it has no medical use. The federal government and still many states would rather throw innocent people in jail for using medical marijuana than be honest about how much it can help people recover from cancer and other diseases.
Below are summaries to just some of the scientific research out there that sustains the belief that medical marijuana will help people cure their cancer.
One of the most exciting areas of current research in the cannabinoid field is the study of the potential application of these compounds as antitumor drugs. CBD represents the first nontoxic exogenous agent that can significantly decrease Id-1 expression in metastatic breast cancer cells leading to the down-regulation of tumor aggressiveness., The CBD concentrations effective at inhibiting Id-1 expression correlated with those used to inhibit the proliferative and invasive phenotype of breast cancer cells. Of the five cannabinoids tested: cannabidiol, cannabigerol, cannnabichromene; cannabidiol-acid and THC-acid, it was found that cannabidiol is the most potent inhibitor of cancer cell growth. Taken together, these data might set the bases for a cannabinoid therapy for the management of breast cancer.
Results show that Δ9-tetrahydrocannabinol reduces tumor growth,
tumor number, and the amount/severity of lung metastases in MMTV-neu
mice. Cannabinoids induce ICAM-1, thereby conferring TIMP-1 induction
and subsequent decreased cancer cell invasiveness thus inhibits lung
cancer invasion and metastasis.
Non-small cell lung cancer (NSCLC) is the leading cause of cancer deaths worldwide. Researchers have observed expression of CB1 (24%) and CB2 (55%) in NSCLC patients. They have also shown that the treatment of NSCLC cell lines (A549 and SW-1573) with CB1/CB2- and CB2-specific agonists Win55,212-2 and JWH-015, respectively, significantly attenuated random as well as growth factor-directed in vitro chemotaxis and chemoinvasion in these cells.
Researchers in lung cancers also reported that they observed
significant reduction in focal adhesion complex, which plays an
important role in cancer migration. Medical marijuana significantly
inhibited in vivo tumor growth and lung metastasis (50%).
In research on pancreatic cancer it was found that cannabinoids
lead to apoptosis of pancreatic tumor cells via a CB2 receptor and de
novo synthesized ceramide-dependent up-regulation of p8 and the
endoplasmic reticulum stress–related genes ATF-4 and TRB3. These
findings may contribute to set the basis for a new therapeutic approach
for the treatment of pancreatic cancer as reported by the National
Prostate cancer cells possess increased expression of both cannabinoid 1 and 2 receptors, and stimulation of these results in decrease in cell viability, increased apoptosis, and decreased androgen receptor expression and prostate-specific antigen excretion.
In colorectal carcinoma cell lines, cannabidiol protected DNA from oxidative damage, increased endocannabinoid levels and reduced cell proliferation in a CB(1)-, TRPV1- and PPARγ-antagonists sensitive manner. It is concluded that cannabidiol exerts chemopreventive effect in vivo and reduces cell proliferation through multiple mechanisms.
Ovarian cancer represents one of the leading cause of cancer-related deaths for women and is the most common gynecologic malignancy. Results with medical marijuana support a new therapeutic approach for the treatment of ovarian cancer. It is also conceivable that with available cannabinoids as lead compounds, non-habit forming agents that have higher biological effects could be developed.
Examination of a number of human leukemia and lymphoma cell lines demonstrate that CB2 cannabinoid receptors expressed on malignancies of the immune system may serve as potential targets for the induction of apoptosis. Also, because CB2 agonists lack psychotropic effects, they may serve as novel anticancer agents to selectively target and kill tumors of immune origin. Plant-derived cannabinoids, including Delta9-tetrahydrocannabinol (THC), induce apoptosis in leukemic cells.
Cannabinoid-treated tumors showed an increased number of apoptotic cells. This was accompanied by impairment of tumor vascularization, as determined by altered blood vessel morphology and decreased expression of proangiogenic factors (VEGF, placental growth factor, and angiopoietin. Abrogation of EGF-R function was also observed in cannabinoid-treated tumors. These results support a new therapeutic approach for the treatment of skin tumors.
Hepatocellular carcinoma (HCC) is the third cause of cancer-related death worldwide. When these tumors are in advanced stages, few therapeutic options are available. In this study, the effects of cannabinoids–a novel family of potential anticancer agents–on the growth of HCC was investigated. It was found that Δ(9)-tetrahydrocannabinol (Δ(9)-THC, the main active component of Cannabis sativa) and JWH-015 (a cannabinoid receptor 2 (CB(2)) cannabinoid receptor-selective agonist) reduced the viability of the human HCC cell lines Cannabinoids were able to inhibit tumor growth and ascites in an orthotropic model of HCC xenograft. These findings may contribute to the design of new therapeutic strategies for the management of HCC.
Both cholangiocarcinoma cell lines and surgical specimens from cholangiocarcinoma patients expressed cannabinoid receptors. THC inhibited cell proliferation, migration and invasion, and induced cell apoptosis. THC also decreased actin polymerization and reduced tumor cell survival in anoikis assay. pMEK1/2 and pAkt demonstrated the lower extent than untreated cells. Consequently, THC is potentially used to retard cholangiocarcinoma cell growth and metastasis.
Smoking marijuana might decrease the smoker’s risk for bladder cancer, a new study shows. Retrospectively analyzing a large database of patients, researchers at Kaiser Permanente in California found that patients who reported cannabis use were 45% less likely to be diagnosed with bladder cancer than patients who did not smoke at all.
THC is a potent inducer of apoptosis, even at 1 x IC(50) (inhibitory concentration 50%) concentrations and as early as 6 hours after exposure to the drug. These effects were seen in leukemic cell lines (CEM, HEL-92, and HL60) as well as in peripheral blood mononuclear cells. Cannabinoids represent a novel class of drugs active in increasing the life span in mice carrying Lewis lung tumors and decreasing primary tumor size.
Research has also found a cannabidiol-driven impaired invasion
of human cervical cancer (HeLa, C33A) and human lung cancer cells (A549)
that was reversed by antagonists to both CB(1) and CB(2) receptors as
well as to transient receptor potential vanilloid 1 (TRPV1). The
decrease of invasion by cannabidiol appeared concomitantly with up
regulation of tissue inhibitor of matrix metalloproteinases-1 (TIMP the
findings provide a novel mechanism underlying the anti-invasive action
of cannabidiol and imply its use as a therapeutic option for the
treatment of highly invasive cancers.
A new anticancer quinone (HU-331) was synthesized from cannabidiol. It shows significant high efficacy against human cancer cell lines in vitro and against in vivo tumor grafts in nude mice. Two non-psychotropic cannabinoids, cannabidiol (CBD) and cannabidiol-dimethylheptyl (CBD-DMH), induced apoptosis in a human acute myeloid leukemia (AML) HL-60 cell line.
Other studies show a synthetic and potent cannabinoid receptor agonist, investigated in hepatoma HepG2 cells and a possible signal transduction pathway that is proposed indicates a potential positive role in liver cancer.Cannabinoids have been found to counteract intestinal inflammation and colon cancer.
The control of the cellular proliferation has become a focus of major attention as opening new therapeutic possibilities for the use of cannabinoids as potential antitumor agents. Cannabinoid treatment inhibits angiogenesis of gliomas in vivo. Remarkably, cannabinoids kill glioma cells selectively and can protect non-transformed glial cells from death. These and other findings reviewed here might set the basis for a potential use of cannabinoids in the management of gliomas. Other confirming studies may provide the basis for a new therapeutic approach for the treatment of malignant gliomas.
Cannabinoids are found to exert their anti-cancer effects in a number of ways and in a variety of tissues.
Triggering cell death, through a mechanism called apoptosis
Stopping cells from dividing
Preventing new blood vessels from growing into tumors
Reducing the chances of cancer cells spreading through the body, by stopping cells from moving or invading neighboring tissue
Speeding up the cell’s internal ‘waste disposal machine’ – a process known as autophagy – which can lead to cell death
All these effects are thought to be caused by cannabinoids locking onto the CB1 and CB2 cannabinoid receptors. Almost daily we are seeing new or confirming evidence that Cannabinoid’s can be used to great benefit in cancer treatment of many types.
What are the other Disease does the cannabis Cures ?
AIDS Related Illness
Amyotrophic Lateral Sclerosis (ALS)
Any Chronic Medical Symptom that Limits Major Life Activities
Arteriosclerotic Heart Disease
Attention Deficit Hyperactivity Disorder (ADD/ADHD)
Brain Tumor, Malignant
Cancer, Adrenal Cortical
Carpal Tunnel Syndrome
Cervical Disk Disease
Chronic Fatigue Syndrome
Chronic renal failure
Damage to Spinal Cord Nervous Tissue
Diabetes, Adult Onset
Diabetes, Insulin Dependent
Diabetic Peripheral Vascular Disease
Inflammatory Autoimmune-mediated Arthritis
Inflammatory Bowel Disease (IBD)
Intermittent Explosive Disorder (IED)
Lou Gehrig’s Disease
Multiple Sclerosis (MS)
Obsessive Compulsive Disorder
Post Polio Syndrome (PPS)
Post-Traumatic Stress Disorder (PTSD)
Premenstrual Syndrome (PMS)
Radiation Therapy (Learn more)
Restless Legs Syndrome (RLS)
Shingles (Herpes Zoster)
Skeletal Muscular Spasticity
Sturge-Weber Syndrome (SWS)
Tardive Dyskinesia (TD)
Temporomandibular Joint Disorder (TMJ)
Keep in mind that the ailments listed above are not necessarily approved by each state as a medically sound reason to receive a medical marijuana recommendation.
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